Overexpression of N-terminal kinase like gene promotes tumorigenicity of hepatocellular carcinoma by regulating cell cycle progression and cell motility

Overexpression of N-terminal kinase like gene promotes tumorigenicity of hepatocellular carcinoma by regulating cell cycle progression and cell motility

10.18632/oncotarget.2730

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書目詳細資料
Main Authors:	Wang, J, Liu, M, Chen, L, Chan, T.H.M, Jiang, L, Yuan, Y.-F, Guan, X.-Y
其他作者:	ANATOMY
格式:	Article
出版:	2020
主題:	agar cyclin D1 dynamin II F actin guanosine triphosphatase n terminal kinase like protein protein protein Cdc42 protein p53 Rac1 protein unclassified drug CHD1L protein, human DNA binding protein DNA helicase DNM2 protein, human dynamin SCYL1 protein, human transcription factor adult amino terminal kinase like gene animal cell animal experiment animal model animal tissue apoptosis Article cancer prognosis carcinogenesis carcinogenicity cell cycle progression cell division cell growth cell motility CHD1L oncogene chromatin immunoprecipitation chromosome segregation colony formation controlled study correlation analysis cytokinesis enzyme phosphorylation female G1 phase cell cycle checkpoint gene gene expression gene overexpression human human tissue lamellipodium liver cell carcinoma major clinical study male mass spectrometry mitosis mouse mutation nonhuman nude mouse oncogene phenotype promoter region real time polymerase chain reaction animal biosynthesis cell cycle cell motion cell proliferation enzymology genetics liver tumor metabolism middle aged pathology physiology tumor cell line xenograft Animals Apoptosis Carcinoma, Hepatocellular Cell Cycle Cell Line, Tumor Cell Movement Cell Proliferation DNA Helicases DNA-Binding Proteins Dynamins Female Gene Expression Heterografts Humans Liver Neoplasms Male Mice Middle Aged Transcription Factors
在線閱讀:	https://scholarbank.nus.edu.sg/handle/10635/180963
標簽:	添加標簽沒有標簽, 成為第一個標記此記錄!
機構:	National University of Singapore

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https://scholarbank.nus.edu.sg/handle/10635/180963