The ALS-FTD-linked gene product, C9orf72, regulates neuronal morphogenesis via autophagy

• المؤلفون الرئيسيون: HO, WAN YUN, TAI, YEE KIT, CHANG, JER-CHERNG, LIANG, JASON, TYAN, SHEUE-HOUY, CHEN, SONG, GUAN, JUN-LIN, ZHOU, HUILIN, SHEN, HAN-MING, KOO, EDWARD, LING, SHUO-CHIEN
• مؤلفون آخرون: MEDICINE
• التنسيق: مقال
• اللغة: English
• منشور في: TAYLOR & FRANCIS INC 2019
• الموضوعات: Science & Technology; Life Sciences & Biomedicine; Cell Biology; Amyotrophic lateral sclerosis (ALS); autophagy; C9orf72; dendritic arborization; frontotemporal dementia (FTD); ULK1; AMYOTROPHIC-LATERAL-SCLEROSIS; REPEAT EXPANSION; ULK1 COMPLEX; PATHOGENIC MECHANISMS; HEXANUCLEOTIDE REPEAT; MUTATIONS; NEURODEGENERATION; DENN; RNA; RAB
• الوصول للمادة أونلاين: https://scholarbank.nus.edu.sg/handle/10635/155081

AUTOPHAGY