Hepatitis C Virus-Induced ROS/JNK Signaling Pathway Activates the E3 Ubiquitin Ligase Itch to Promote the Release of HCV Particles via Polyubiquitylation of VPS4A

• المؤلفون الرئيسيون: Deng, Lin, Liang, Yujiao, Ariffianto, Adi, Matsui, Chieko, Abe, Takayuki, Muramatsu, Masamichi, Wakita, Takaji, Maki, Masatoshi, Shibata, Hideki, Shoji, Ikuo
• التنسيق: مقال PeerReviewed
• اللغة: English
• منشور في: American Society for Microbiology 2022
• الموضوعات: Medical Biochemistry and Metabolomics; Infectious Diseases
• الوصول للمادة أونلاين: https://repository.ugm.ac.id/279044/1/Hepatitis%20C%20Virus-Induced%20ROS%20%20JNK%20Signaling%20Pathway%20Activates%20the%20E3%20Ubiquitin%20Ligase%20Itch%20to%20Promote%20the%20Release%20of%20HCV%20Particles%20via%20Polyubiquitylation%20of%20VPS4A.pdf; https://repository.ugm.ac.id/279044/; https://journals.asm.org/doi/10.1128/jvi.01811-21; https://doi.org/10.1128/jvi.01811-21
• المؤسسة: Universitas Gadjah Mada

We previously reported that hepatitis C virus (HCV) infection activates the reactive oxygen species (ROS)/c-Jun N-terminal kinase (JNK) signaling pathway. However, the roles of ROS/JNK activation in the HCV life cycle remain unclear. We sought to identify a novel role of the ROS/JNK signaling pathwa...