Nrf2 is a central regulator of metabolic reprogramming of myeloid-derived suppressor cells in steady state and sepsis

10.3389/fimmu.2018.01552

محفوظ في:

التفاصيل البيبلوغرافية
المؤلفون الرئيسيون:	Ohl, K, Fragoulis, A, Klemm, P, Baumeister, J, Klock, W, Verjans, E, Böll, S, Möllmann, J, Lehrke, M, Costa, I, Denecke, B, Schippers, A, Roth, J, Wagner, N, Wruck, C, Tenbrock, K
مؤلفون آخرون:	SAW SWEE HOCK SCHOOL OF PUBLIC HEALTH
التنسيق:	مقال
منشور في:	2020
الموضوعات:	CD11b antigen CD4 antigen complementary DNA deoxyribonuclease I granulocyte macrophage colony stimulating factor inducible nitric oxide synthase interleukin 1 interleukin 12 interleukin 6 kelch like ECH associated protein 1 toll like receptor 4 transcription factor Nrf2 transcriptome uracil DNA glycosidase aerobic glycolysis animal cell animal experiment animal model animal tissue Article CD4+ T lymphocyte cell cycle cell expansion colitis controlled study gene expression immunological tolerance immunomodulation in vitro study in vivo study lipopolysaccharide-induced sepsis metabolic activation metabolic regulation mitochondrial respiration mouse myeloid-derived suppressor cell nonhuman nuclear reprogramming oxidative stress transcriptomics
الوصول للمادة أونلاين:	https://scholarbank.nus.edu.sg/handle/10635/178079
الوسوم:	إضافة وسم لا توجد وسوم, كن أول من يضع وسما على هذه التسجيلة!
المؤسسة:	National University of Singapore

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